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Vets With Traumatic Brain Injury Have Higher Suicide Risk: Study

WEDNESDAY, Sept. 11, 2019 — The risk of suicide among U.S. military veterans who have suffered a traumatic brain injury (TBI) is more than double that of other vets, a new study finds.

Researchers analyzed records of more than 1.4 million vets who received care from the U.S. Veterans Health Administration (VHA) between 2005 and 2015.

They compared severity of the traumatic brain injury with diagnoses of psychiatric and other medical conditions, along with method of death in suicide cases.

After adjusting for depression and other mental health diagnoses, the researchers found that veterans with moderate or severe TBI were 2.45 times more likely to die by suicide than those without TBI. The study only found an association and not a cause-and-effect link, however.

During the study period, the suicide rate was 86 per 100,000 person years for those with TBI and 37 per 100,000 person years for others. A person year is a formula that accounts for the number of people in a study and how long they were followed.

Of vets studied who died by suicide, 68% used guns. The rate was 78% among vets with moderate or severe TBI, according to the University of Colorado-led study.

“Together, these findings underscore the importance of understanding veterans’ lifetime history of TBI to prevent future deaths by suicide, and support the implementation of screening initiatives for lifetime history of TBI among all individuals utilizing the VHA,” the authors said in a university news release.

Lisa Brenner, a professor of physical medicine and rehabilitation at the University of Colorado School of Medicine, led the study.

Her team said the findings also show the need for more research regarding gun safety among those with moderate to severe TBI.

The study was published recently in the Journal of Head Trauma Rehabilitation.

More information

The U.S. National Institute of Neurological Disorders and Stroke has more on traumatic brain injury.

© 2019 HealthDay. All rights reserved.

Posted: September 2019

Drugs.com – Daily MedNews

Surgeon General: Marijuana Bad for Developing Brain

August 29, 2019 — Marijuana use is risky for young people and pregnant women, a U.S. Surgeon General health advisory warns.

The latest research shows that marijuana is particularly harmful to developing brains and can be passed along to infants in the womb or through breast milk, U.S. Surgeon General Jerome Adams said Thursday, ABC News reported.

Adams said he was deeply concerned about what he called the “rapid normalization” of marijuana and the mistaken belief among young people that because the drug is now legal in some states, it must be safe.

“Not enough people known that today’s marijuana is far more potent than in days past,” Adams said, ABC News reported. “This ain’t your mother’s marijuana,” he added.

The American Academy of Pediatrics has long advised young people not to use marijuana and has said that no amount of marijuana has been proven safe to use while pregnant or breast feeding.

Adams and Health Human Services Secretary Alex Azar said they support additional research on the effects of marijuana, and Azar said the federal government will launch a public awareness campaign, ABC News reported.

Last year, the surgeon general issued a warning about e-cigarette use by young people, called it “unsafe” in any form.

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Mosquito-Borne Brain Infection Found in Florida

July 29, 2019 — There’s an increased risk of a mosquito-borne virus that causes brain infection and swelling, Florida health officials warn.

Eastern equine encephalitis (EEE) has been detected in several sentinel chickens, according to the Florida Department of Health in Orange County, CNN reported.

Sentinel chickens are tested regularly for the West Nile virus and EEE.

After the positive tests for EEE virus in the sentinel chickens in Orange Couty, the health department said “the risk of transmission to humans has increased,” CNN reported.

Only about seven human cases of the EEE virus reported in the US each year. However, about one-third of people who contract it die, and many survivors have long-term neurological problems, according to the Centers for Disease Control and Prevention.

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More Evidence That Socializing Helps Protect the Aging Brain

MONDAY, July 15, 2019 — Join a book club, take a cruise or just visit friends — new research supports the notion that social activities help stave off mental decline as you age.

The study found that seniors with high levels of an Alzheimer’s-linked protein in their brains were able to slow any mental decline if they got out and socialized regularly.

So, “social engagement may be an important marker of resilience” in older adults at risk of dementia, said senior author Dr. Nancy Donovan. She’s chief of geriatric psychiatry at Brigham and Women’s Hospital in Boston.

In the study, researchers tracked data on 217 men and women aged 63 to 89. These seniors were all taking part in the Harvard Aging Brain Study, a trial aimed at identifying early signs of Alzheimer’s disease.

Participants started the study with no evidence of mental decline, but some had high levels of amyloid beta protein, a hallmark of Alzheimer’s that can be detected in brain scans.

The researchers assessed seniors’ levels of social engagement (such as spending time with friends and family, and doing volunteer work) and their mental (cognitive) function at the start of the study and again three years later.

Among seniors with high levels of amyloid beta, those with lower levels of social engagement at the start had greater mental decline after three years than those who were socially active, the findings showed.

This association was not seen among people with low levels of amyloid beta, according to the study, which was recently published in the American Journal of Geriatric Psychiatry.

“Social engagement and cognitive function are related to one another and appear to decline together,” Donovan said in a hospital news release.

Her team believes longer studies might add more insight into mental decline over time as well as Alzheimer’s progression.

“We want to understand the breadth of this issue in older people and how to intervene to protect high-risk individuals and preserve their health and well-being,” Donovan explained.

This study relied on a standard measure of social engagement that didn’t assess all the subtle effects of digital communication or all the impacts of relationships, the researchers noted.

A more comprehensive assessment could be valuable in future clinical trials of Alzheimer’s disease, the authors added.

More information

The U.S. National Institute on Aging has more about Alzheimer’s disease.

© 2019 HealthDay. All rights reserved.

Posted: July 2019

Drugs.com – Daily MedNews

Brain Games: Mental Stimulation Keeps Dogs’ Minds Sharp

By Jodi Helmer

man playing with dog

Long walks and romps at the dog park keep your dog in top physical shape, but what about her mental health? Like their owners, dogs can have mental decline, says Leticia Fanucchi, DVM, a veterinarian and clinical instructor at Washington State University in Pullman, WA. “Dogs shouldn’t stop being mentally and physically active just because they get older,” she says.

A decline in thinking skills often affects dogs older than 8 years. Signs of canine mental decline include disorientation, changes in sleep/wake cycles, housetraining accidents, higher anxiety, and less desire for physical activity and social interaction. Dogs that have memory loss may have forgotten routine commands such as “sit” and “stay.”

Medications can help slow this decline, but a 2018 study found that mental stimulation could also improve brain health. Researchers at Messerli Research Institute in Vienna used a touchscreen to teach dogs simple computer games; the dogs received treats for getting the answers correct.

“These kinds of mental games help wake up areas of the brain that have been inactive,” Fanucchi says. The combination of sight, scent, and spatial orientation required to solve the puzzle helped make connections between different parts of the brain. The tasty reward motivated the dogs to stick with the activity.

While the touchscreen games used in lab research aren’t sold in stores (yet), Fanucchi suggests creating your own brain games at home. Offer your dog interactive toys that require her to move a puzzle piece or roll a cube to release a treat. And get your pet outside. Regular walks allow her to explore new sights, sounds, and smells, which can be mentally stimulating, and keeping her active during daylight hours can also help reset a confused sleep/wake cycle.

Don’t wait until your pet starts showing signs of doggie dementia to introduce brain training. “Mental stimulation benefits dogs of all ages and is especially important for older dogs,” Fanucchi says.

4 Questions

Worried about your older dog’s brain health? “Tell your vet about recent changes in your dog’s behavior,” says Fanucchi, and consider these questions.

Does your dog show signs of mental decline? Don’t assume that ignoring commands and having accidents in the house are signs of a defiant dog.

Could other health issues be causing these symptoms? Before diagnosing mental decline, Fanucchi says your vet will want to rule out endocrine issues, heart disease, and other medical conditions that could cause similar symptoms.

Is medication available? There is a medication approved to treat mental decline in dogs. Fanucchi suggests talking to your veterinarian about whether it’s right for your pet.

Will changes in diet help? Research shows that kibble made for senior dogs could help improve mental skills and slow decline. Ask about brain protection blends that contain triglycerides, B vitamins, and antioxidants.

Find more articles, browse back issues, and read the current issue of WebMD Magazine.

Sources

SOURCES:

Leticia Fanucchi, DVM, Washington State University, Pullman, WA.

Chapagain, D. Gerontology, February 2018.

Wallace, L. Proceedings of the Fourth International Conference on Animal-Computer Interaction, November 2018.

Szabo, D. Behavioural Processes, December 2018.

Pan, Y. Frontiers in Nutrition, December 2018.

© 2019 WebMD, LLC. All rights reserved.

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Education, Intelligence Might Protect Your Brain

FRIDAY, June 14, 2019 — Being smart and highly educated may not prevent Alzheimer’s disease, but it appears to delay the disease’s impact on everyday life, a new study suggests.

Researchers can’t prove that that’s the case, but their data suggests it might be.

“Our study was designed to look for trends, not prove cause and effect, but the major implication of our study is that exposure to education and better cognitive performance when you’re younger can help preserve cognitive function for a while, even if it’s unlikely to change the course of the disease,” said study author Dr. Rebecca Gottesman. She’s a professor of neurology at the Johns Hopkins University School of Medicine in Baltimore.

For the study, Gottesman and her team collected data on 331 middle-aged and older people without dementia who were followed for 20 years and had brain scans as part of a separate study. The scans revealed how much plaques were in their brains, a hallmark of Alzheimer’s. The group included people with less than a high school education and those who went to college.

The researchers found that those with more education scored better on tests of memory and language than those with less education, no matter how much plaque their brains contained.

They also found that cognition scores in midlife did not affect the amounts of plaque found later in life.

“Our data suggest that more education seems to play a role as a form of cognitive reserve that helps people do better at baseline, but it doesn’t affect one’s actual level of decline,” Gottesman said in a university news release.

The report was published in the April issue of the Journal of Alzheimer’s Disease.

More information

For more about Alzheimer’s disease, visit the Alzheimer’s Association.

© 2019 HealthDay. All rights reserved.

Posted: June 2019

Drugs.com – Daily MedNews

Brain Zoo Studios Working on Animated Mueller Report

L.A.-based Brain Zoo Studios and Julian August Productions are working on The Mueller Report: An Animated Series. The topical animated TV series hopes to make the report’s findings more accessible to the public. In the coming weeks, the partnership will launch a crowd-funding campaign through Kickstarter.

“Erick and I have received much positive feedback on the project. The report is a long legal document, and people have busy lives. We believe combining education and entertainment is the best way to communicate what is in the report to the general public.” said, Mo Davoudian, the Emmy-winning producer at Brain Zoo Studios and Julian August Productions.

“Mo and I feel it’s our patriotic duty to make this series and we believe everyone needs to know what is in the report. We have found others who feel this project is important, including Emmy-nominated voice actor John Di Domenico who will be the voice of Trump.” said, Erick Armelin producer at Brain Zoo Studios and Julian August Productions.

Actor John Di Domenico has been seen and heard as Trump since 2004 on
television, feature films, podcasts, web series, apps, games and hundreds of live shows around the world. For more info visit www.brainzoostudios.com

The Mueller Report: An Animated Series

The Mueller Report: An Animated Series

Animation Magazine

New Brain Cells Grow Later In Life Than We Think

By Robert Preidt

HealthDay Reporter

WEDNESDAY, May 29, 2019 (HealthDay News) — New research delivers fresh hope for everyone who struggles with a fading memory: Neurons continue to form well into old age, even in people with mental impairments or Alzheimer’s disease.

“We found that there was active neurogenesis [new neurons forming] in the hippocampus of older adults well into their 90s,” said study author Orly Lazarov, a professor of anatomy and cell biology at the University of Illinois at Chicago.

“The interesting thing is that we also saw some new neurons in the brains of people with Alzheimer’s disease and cognitive [thinking] impairment,” she added in a university news release.

The findings could lead to new treatments for mental decline in older adults, the researchers said.

In the study, Lazarov and her colleagues examined hippocampus tissue from the brains of 18 people, average age 90.6 years, after they died.

The hippocampus is involved in the formation of memories and in learning.

On average, there were about 2,000 neural stem cells and 150,000 developing neurons in each brain.

While people with mental impairments and Alzheimer’s disease did have new neurons, their levels were significantly lower than in people with normal brain function, the researchers noted.

This is the first evidence of significant numbers of neural stem cells and newly developing neurons in the hippocampus of elderly adults, even in those with disorders that affect that part of the brain.

The researchers also found that people who scored better on tests of mental skills had more newly developing neurons in the hippocampus than those who scored lower on the tests, regardless of the level of disease in the brain.

“The mix of the effects of pathology and neurogenesis is complex and we don’t understand exactly how the two interconnect, but there is clearly a lot of variation from individual to individual,” Lazarov said.

“The fact that we found that neural stem cells and new neurons are present in the hippocampus of older adults means that if we can find a way to enhance neurogenesis, through a small molecule, for example, we may be able to slow or prevent cognitive decline in older adults, especially when it starts, which is when interventions can be most effective,” she said.

The findings were published May 23 in the journal Cell Stem Cell.

WebMD News from HealthDay

Sources

SOURCE: University of Illinois at Chicago, news release, May 24, 2019

Copyright © 2013-2018 HealthDay. All rights reserved.

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Could Alzheimer’s Spread Like Infection Throughout the Brain?

WEDNESDAY, May 1, 2019 — With findings that might alter the path of Alzheimer’s research, scientists say misfolded forms of two proteins appear to spread through patients’ brains similar to an infection.

The findings suggest that Alzheimer’s is a “double-prion” disorder. This discovery could help lead to new treatments that focus directly on prions, according to researchers from the University of California, San Francisco.

A prion is a misshapen protein that can force other copies of that protein into the same misfolded shape and spread in the brain. It’s best known for its role in bovine spongiform encephalopathy — “mad cow” disease — and Creutzfeldt-Jakob disease (CJD), a degenerative brain disorder.

In the new research, the university team analyzed the brains of 75 Alzheimer’s patients after death and found self-propagating prion forms of the proteins amyloid beta and tau. Higher amounts of these prions were associated with early-onset Alzheimer’s and younger age at death.

Alzheimer’s patients have amyloid plaques and tau tangles in the brain, but efforts to treat the disease by clearing out these inactive proteins have failed.

These new findings suggest that active amyloid beta and tau prions could drive Alzheimer’s and offer targets for effective treatment, according to the researchers.

“I believe this shows beyond a shadow of a doubt that amyloid beta and tau are both prions, and that Alzheimer’s disease is a double-prion disorder in which these two rogue proteins together destroy the brain,” said study senior author Dr. Stanley Prusiner, director of the UCSF Institute for Neurodegenerative Diseases. Prusiner won a Nobel Prize in 1997 for discovering that prions were responsible for mad cow disease and CJD.

Prion levels also appear linked to patient longevity, he noted.

“We need a sea change in Alzheimer’s disease research, and that is what this paper does. This paper might catalyze a major change in AD research,” Prusiner said in a university news release.

For this study, the researchers used recently developed laboratory tests to rapidly measure prions in human tissue samples. They can reveal infectious prion levels in just days.

These tests “are a game-changer,” said study co-author William DeGrado, a UCSF professor of pharmaceutical chemistry.

In order to develop effective therapies and diagnostics, scientists must target the active prion forms, rather than the large amount of protein in plaques and tangles, DeGrado said.

The researchers hope that measuring the prion forms of amyloid beta and tau might lead to the development of drugs that either prevent them from forming or spreading, or help remove them before they cause damage.

The study was published May 1 in the journal Science Translational Medicine.

More information

The U.S. National Institute of Neurological Disorders and Stroke has more on Alzheimer’s disease.

© 2019 HealthDay. All rights reserved.

Posted: May 2019

Drugs.com – Daily MedNews

Brain Scans Spot, Track Alzheimer’s

By Robert Preidt

HealthDay Reporter

TUESDAY, April 2, 2019 (HealthDay News) — Brain scans can improve diagnosis and management of Alzheimer’s disease, a new study claims.

Researchers assessed the use of PET scans to identify Alzheimer’s-related amyloid plaques in the brain. The study included more than 11,000 Medicare beneficiaries with mild thinking impairment or dementia of uncertain cause.

This scanning technique changed the diagnosis of the cause of mental impairment in more than one-third of the participants in the study.

The brain scan results also changed management — including the use of medications and counseling — in nearly two-thirds of cases, according to the study published April 2 in the Journal of the American Medical Association.

“These results present highly credible, large-scale evidence that amyloid PET imaging can be a powerful tool to improve the accuracy of Alzheimer’s diagnosis and lead to better medical management, especially in difficult-to-diagnose cases,” said study co-author Maria Carrillo, chief science officer of the Alzheimer’s Association.

“It is important that amyloid PET imaging be more broadly accessible to those who need it,” she added in an association news release.

Funding for the study came from Avid Radiopharmaceuticals Inc., General Electric Healthcare, and Life Molecular Imaging.

“We are impressed by the magnitude of these results, which make it clear that amyloid PET imaging can have a major impact on how we diagnose and care for patients with Alzheimer’s disease and other forms of cognitive decline,” said lead author Dr. Gil Rabinovici. He’s a professor of neurology at the Memory and Aging Center at the University of California, San Francisco.

There is no cure for Alzheimer’s disease, but early diagnosis means that patients can receive treatment to manage symptoms and be directed to clinical trials for new drugs.

Early diagnosis also means that patients and families can plan for the future, including safety, care, legal and financial issues, and access resources and support programs, the researchers said.

In this study, the PET scans revealed that about one-third of patients previously diagnosed with Alzheimer’s had no significant amyloid buildup, and their Alzheimer’s diagnosis was reversed.

Continued

But in nearly half of patients not previously diagnosed with Alzheimer’s, the PET scans revealed significant amyloid plaque buildup, resulting in a new diagnosis of Alzheimer’s.

One-third of the study participants who had previously been referred to Alzheimer’s clinical trials showed no sign of amyloid buildup based on PET scans. Based on those results, doctors were able to ensure that nearly all (93 percent) of patients referred to Alzheimer’s trials were amyloid-positive, which is critical to these trials’ success.

“Accurate diagnoses are critical to ensure patients are receiving the most appropriate treatments. In particular, Alzheimer’s medications can worsen cognitive decline in people with other brain diseases,” Rabinovici said.

“But perhaps more fundamentally, people who come into the clinic with concerns about memory problems want answers. An early, definitive diagnosis may allow individuals to be part of planning for the next phase of their lives and to make decisions that otherwise would eventually need to be made by others,” he said.

WebMD News from HealthDay

Sources

SOURCE: Alzheimer’s Association, news release, April 2, 2019

Copyright © 2013-2018 HealthDay. All rights reserved.

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Pagination

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Coffee on Your Mind? Even Thinking About It ‘Arouses’ the Brain

MONDAY, April 1, 2019 — Need a quick pick-me-up? Just thinking about a cup of joe can give you a mental boost, researchers say.

“Coffee is one of the most popular beverages and a lot is known about its physical effects,” said study co-author Sam Maglio, associate professor of management at the UNi. “Much less is known about its psychological meaning — in other words, how even seeing reminders of it can influence how we think.”

But Maglio said just looking at things that call coffee to mind can arouse a java junkie’s brain. It’s all due to what he called priming, in which exposure to cues about something can affect thoughts and behavior.

“People often encounter coffee-related cues, or think about coffee, without actually ingesting it,” Maglio said in a university news release. “We wanted to see if there was an association between coffee and arousal such that if we simply exposed people to coffee-related cues, their physiological arousal would increase, as it would if they had actually drank coffee.”

Arousal refers to the way specific brain areas get activated into a state of being alert, awake and attentive, according to the researchers. The trigger can be emotions, neurotransmitters in the brain — and even the caffeinated beverages we favor.

To check it out, Maglio and fellow University of Toronto researcher Eugene Chan compared coffee- and tea-related cues among participants from Western and Eastern cultures.

Those exposed to coffee-related cues perceived time as shorter and thought in more concrete, precise terms. Subjective and physiological arousal probably explains these effects, according to the study.

But everyone didn’t get the same boost. Coffee-related cues did not provoke as much arousal for participants from Eastern cultures, probably because they are not coffee-dominated cultures, Maglio said.

“In North America we have this image of a prototypical executive rushing off to an important meeting with a triple espresso in their hand. There’s this connection between drinking caffeine and arousal that may not exist in other cultures,” he said.

The study was published in the April issue of the journal Consciousness and Cognition.

Next up: Researchers will examine associations people have for different foods and beverages.

More information

The American Heart Association looks at coffee and your health.

© 2019 HealthDay. All rights reserved.

Posted: April 2019

Drugs.com – Daily MedNews

Only Spoken Words Processed in Newly Discovered Brain Region

WEDNESDAY, March 27, 2019 — A dementia study has led researchers to a brain region that processes spoken, not written, words.

Northwestern University researchers worked with four patients who had a rare type of dementia called primary progressive aphasia (PPA), which destroys language.

Although able to hear and speak, they could not understand what was said out loud. However, they could still process written words. For example, if they read the word “hippopotamus,” they could identify a picture of a hippo. But if someone said the word “hippopotamus,” they couldn’t point to its picture.

Through their tests with these patients, the researchers were able to identify an area in the left brain that appears specialized to process spoken words.

“We always think of these degenerative diseases as causing widespread impairment, but in early stages, we’re learning that neurodegenerative disease can be selective with which areas of the brain it attacks,” said senior author Sandra Weintraub. She’s a professor of psychiatry and behavioral sciences and neurology at Northwestern’s Feinberg School of Medicine in Chicago.

“The fact that only the auditory words were impaired in these patients and their visual words were untouched leads us to believe we’ve identified a new area of the brain where raw sound information is transformed into auditory word images,” Weintraub explained in a university news release.

Because the study included only four patients, the findings are preliminary. But the study authors said further research could improve understanding of this type of dementia and lead to therapies for it that focus on written, rather than spoken, communication.

“It’s typically very frustrating for patients with PPA and their families,” said Weintraub. “The person looks fine, they’re not limping and yet they’re a different person. It means having to readjust to this person and learning new ways to communicate.”

The study was published March 21 in the journal Cognitive and Behavioral Neurology.

More information

The National Aphasia Association has more on primary progressive aphasia.

© 2019 HealthDay. All rights reserved.

Posted: March 2019

Drugs.com – Daily MedNews

Does Diabetes Damage Brain Health?

By Serena Gordon

HealthDay Reporter

FRIDAY, Dec. 14, 2018 (HealthDay News) — Diabetes has been tied to a number of complications such as kidney disease, but new research has found that older people with type 2 diabetes can also have more difficulties with thinking and memory.

During a five-year study, participants with diabetes showed a decline in verbal memory and fluency. Using MRI scans, researchers saw that the participants’ brains were smaller at the start of the study — but the rates of decline in brain size did not differ over the years the patients were followed. The investigators didn’t find a connection between brain size and the thinking and memory troubles.

“Although memory and executive function [thinking and planning skills] declined at a greater rate in people with type 2 diabetes, this was not explained by a decline in brain volume,” said study author Michele Callisaya, a research fellow at the University of Tasmania.

Callisaya said the researchers were surprised by this finding. They expected that decreased brain volume would have been more common in people who were having memory and thinking issues. But she added that it’s possible over a longer time, a relationship between these factors might become evident.

And, she added, “The overall message is that type 2 diabetes affects brain function.”

Past research has found that having diabetes might double a person’s risk of dementia, the researchers said. Although previous studies have shown the connection between the two conditions, none has proven a cause-and-effect relationship. That’s what prompted Callisaya and her colleagues to look at whether or not a loss of brain volume might be behind the connection.

They recruited more than 700 people between 55 and 90 years old for the study. At three different points during the five years, the participants underwent testing to measure their thinking, planning and memory skills. They also had an MRI scan each time.

About half of the participants had type 2 diabetes (348 people) and their average age was 68. The group without diabetes had an average age of 73.

Continued

The researchers found that people with diabetes had lower scores on verbal memory and verbal fluency tests.

Verbal memory is the ability to recall words, and verbal fluency is a measure of thinking and planning skills. People who have problems in these areas might forget people’s names or have trouble finding things more frequently, Callisaya said. People who have trouble in verbal fluency might have difficulties with planning, initiating and organizing things, she added.

The MRI scans showed that people with diabetes had smaller brain volume at the start of the study than people without the blood sugar disorder. But Callisaya’s team saw no evidence that brain size was directly related to the declines in thinking and memory.

Dr. Gisele Wolf-Klein, director of geriatric education at Northwell Health in Great Neck, N.Y., reviewed the findings and said, “While there is no doubt that diabetes is a risk factor for the development of cognitive changes, the relationship with brain atrophy remains uncertain.”

Dr. Joel Zonszein, director of the clinical diabetes center at Montefiore Medical Center in New York City, agreed the study didn’t show any correlation between brain size, diabetes and thinking and memory troubles.

Zonszein said the differences in the two study groups may have played a significant role in the study’s findings. He said the people in the diabetes group were heavier, and had higher cholesterol and blood pressure than people in the other group.

“The take-home message to me is that good early control of all of these risk factors — blood sugar, cholesterol, weight and blood pressure — is important, along with getting good, regular exercise. People who have these risk factors have a higher risk for cognitive decline,” he said.

Wolf-Klein said that, while it hasn’t been proven that good blood sugar management can reduce the risk of brain health issues, “physical activity and a wholesome diet have been associated with a lesser risk of dementia in the general population, as well as a decreased incidence of diabetes.”

Callisaya agreed. “What’s good for the heart is also good for the brain,” she said. In addition to a healthy diet and regular activity, she also recommends to stay social and to keep challenging your brain.

The study was published Dec. 13 in the journal Diabetologia.

WebMD News from HealthDay

Sources

SOURCES: Michele Callisaya, Ph.D., research fellow, University of Tasmania, and adjunct senior lecturer, Monash University, Melbourne, Australia; Gisele Wolf-Klein, M.D., director, geriatric education, Northwell Health, Great Neck, N.Y.; Joel Zonszein, M.D., director, clinical diabetes center, Montefiore Medical Center, New York City; Dec. 13, 2018,Diabetologia

Copyright © 2013-2018 HealthDay. All rights reserved.

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